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NTIS 바로가기Cellular and molecular gastroenterology and hepatology, v.5 no.4, 2018년, pp.678 - 690.e1
Bockerstett, Kevin A. (Department of Molecular Microbiology and Immunology, Saint Louis University School of Medicine, St Louis, Missouri) , Osaki, Luciana H. (Division of Gastroenterology, Departments of Medicine, Pathology and Immunology, Developmental Biology, Washington University School of Medicine, St Louis, Missouri) , Petersen, Christine P. (Nashville VA Medical Center and Departments of Surgery and Cell and Developmental Biology, Epithelial Biology Center, Vanderbilt University School of Medicine, Nashville, Tennessee) , Cai, Catherine W. (Department of Molecular Microbiology and Immunology, Saint Louis University School of Medicine, St Louis, Missouri) , Wong, Chun Fung (Department of Molecular Microbiology and Immunology, Saint Louis University School of Medicine, St Louis, Missouri) , Nguyen, Thanh-Long M. (Department of Molecular Microbiology and Immunology, Saint Louis University School of Medicine, St Louis, Missouri) , Ford, Eric L. (Department of Molecular Microbiology and Immunology, Saint Louis University School of Medicine, St Louis, Missouri) , Hoft, Daniel F. (Department of Molecular Microbiology and Immunology, Saint Louis) , Mills, Jason C. , Goldenring, James R. , DiPaolo, Richard J.
Background & AimsAtrophic gastritis caused by chronic inflammation in the gastric mucosa leads to the loss of gastric glandular cells, including acid-secreting parietal cells. Parietal cell atrophy in a setting of chronic inflammation induces spasmolytic polypeptide expressing metaplasia, a critical...
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