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The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology, v.17 no.1, 2013년, pp.57 - 64
Bae, Yeon Ju (Department of Physiology, Institute of Health Sciences and Medical Research Center for Neural Dysfunction, Gyeongsang National University School of Medicine) , Yoo, Jae-Cheal (Department of Physiology, Institute of Health Sciences and Medical Research Center for Neural Dysfunction, Gyeongsang National University School of Medicine) , Park, Nammi (Department of Physiology, Institute of Health Sciences and Medical Research Center for Neural Dysfunction, Gyeongsang National University School of Medicine) , Kang, Dawon (Department of Physiology, Institute of Health Sciences and Medical Research Center for Neural Dysfunction, Gyeongsang National University School of Medicine) , Han, Jaehee (Department of Physiology, Institute of Health Sciences and Medical Research Center for Neural Dysfunction, Gyeongsang National University School of Medicine) , Hwang, Eunmi (Department of Physiology, Institute of Health Sciences and Medical Research Center for Neural Dysfunction, Gyeongsang National University School of Medicine) , Park, Jae-Yong (Department of Physiology, Institute of Health) , Hong, Seong-Geun
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Cells can resist and even recover from stress induced by acute hypoxia, whereas chronic hypoxia often leads to irreversible damage and eventually death. Although little is known about the response(s) to acute hypoxia in neuronal cells, alterations in ion channel activity could be preferential. This ...
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