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Immunity, v.6 no.2, 1997년, pp.199 - 208
Ikeda, Hideyuki (Cellular Genetics Unit, Université) , Lethé, Bernard (Catholique de Louvain, and Ludwig Institute for Cancer Research, Brussels Branch, 74 avenue Hippocrate, B-1200 Brussels, Belgium) , Lehmann, Frédéric (Cellular Genetics Unit, Université) , Van Baren, Nicolas (Catholique de Louvain, and Ludwig Institute for Cancer Research, Brussels Branch, 74 avenue Hippocrate, B-1200 Brussels, Belgium) , Baurain, Jean-François (Cellular Genetics Unit, Université) , De Smet, Charles (Catholique de Louvain, and Ludwig Institute for Cancer Research, Brussels Branch, 74 avenue Hippocrate, B-1200 Brussels, Belgium) , Chambost, Hervé (Cellular Genetics Unit, Université) , Vitale, Massimo (Catholique de Louvain, and Ludwig Institute for Cancer Research, Brussels Branch, 74 avenue Hippocrate, B-1200 Brussels, Belgium) , Moretta, Alessandro (Cellular Genetics Unit, Université) , Boon, Thierry (Catholique de Louvain, and Ludwig Institute for Cancer Research, Brussels Branch, 74 avenue Hippocrate, B-1200 Brussels, Belgium) , Coulie, Pierre G (Cellular Genetics Unit, Université)
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AbstractMelanoma lines MEL.A and MEL.B were derived from metastases removed from patient LB33 in 1988 and 1993, respectively. The MEL.A cells express several antigens recognized by autologous cytolytic T lymphocytes (CTL) on HLA class I molecules. The MEL.B cells have lost expression of all class I ...
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