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Immunity, v.11 no.5, 1999년, pp.591 - 601
Natarajan, Kannan (Molecular Biology Section, Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, Bethesda, Maryland 20892, USA) , Boyd, Lisa F (Molecular Biology Section, Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, Bethesda, Maryland 20892, USA) , Schuck, Peter (Molecular Interactions Resource, Bioengineering and Physical Science Program, National Institutes of Health, Bethesda, Maryland 20892, USA) , Yokoyama, Wayne M (Division of Rheumatology, Howard Hughes Medical Institute, Washingt.on University School of Medicine, St. Louis, Missouri 63110, USA) , Eilat, Dan (Division of Medicine, Hadassah University Hospital, Jerusalem 91120, Israel) , Margulies, David H (Molecular Biology Section, Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, Bethesda, Maryland 20892, USA)
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AbstractNatural killer cell function is controlled by interaction of NK receptors with MHC I molecules expressed on target cells. We describe the binding of bacterially expressed Ly49A, the prototype murine NK inhibitory receptor, to similarly engineered H-2Dd. Despite its homology to C-type lectins...
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