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NTIS 바로가기Neuron, v.25 no.3, 2000년, pp.537 - 547
Marek, Kurt W. (Department of Biochemistry and Biophysics, Programs in Cell Biology and Neuroscience, University of California Medical School, San Francisco, California 94143, USA) , Ng, Norman (Department of Biochemistry and Biophysics, Programs in Cell Biology and Neuroscience, University of California Medical School, San Francisco, California 94143, USA) , Fetter, Richard (Department of Molecular and Cell Biology, Howard Hughes Medical Institute, University of California, Berkeley, Berkeley, California 94720, USA) , Smolik, Sarah (Department of Cell and Developmental Biology, Oregon Health Science University, Portland, Oregon 97201, USA) , Goodman, Corey S. (Department of Molecular and Cell Biology, Howard Hughes Medical Institute, University of California, Berkeley, Berkeley, California 94720, USA) , Davis, Graeme W. (Department of Biochemistry and Biophysics, Programs in Cell Biology and Neuroscience, University of California Medical School, San Francisco, California 94143, USA)
AbstractPostsynaptic dCBP (Drosophila homolog of the CREB binding protein) is required for presynaptic functional development. Viable, hypomorphic dCBP mutations have a ∼50% reduction in presynaptic transmitter release without altering the Ca2+ cooperativity of release or synaptic ultrastruct...
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