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NTIS 바로가기Muscle & nerve, v.23 no.12, 2000년, pp.1856 - 1861
Karandreas, Nikos (EMG Laboratory, Department of Neurology, Aeginition Hospital, University of Athens, Vas. Sofias 72-74, 11528 Athens, Greece) , Stathis, Pantelis (Department of Neurology, 1st Hospital of Social Security Services, Athens, Greece) , Anastasakis, Aris (Department of Cardiology, University of Athens, Athens, Greece) , Rigopoulos, Angelos (Department of Cardiology, University of Athens, Athens, Greece) , Piperos, Panayiotis (EMG Laboratory, Department of Neurology, Aeginition Hospital, University of Athens, Vas. Sofias 72-74, 11528 Athens, Greece) , Theopistou, Artemis (Department of Cardiology, University of Athens, Athens, Greece) , Stefanadis, Chris (Department of Cardiology, University of Athens, Athens, Greece) , Toutouzas, Pavlos (Department of Cardiology, University of Athens, Athens, Greece)
Hypertrophic cardiomyopathy (HCM) is due to a number of mutations of contractile protein genes such as β-cardiac myosin, myosin binding protein-C, and troponin-T. Unlike troponin-T, β-myosin is a constituent of slow skeletal muscle and its mutations generally have a better prognosis. In or...
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