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[해외논문] Increased neuronal damage and apoE immunoreactivity in human apolipoprotein E, E4 isoform‐specific, transgenic mice after global cerebral ischaemia

The European journal of neuroscience, v.12 no.12, 2000년, pp.4309 - 4317  

Horsburgh, Karen (1 Department of Neuropathology, University of Glasgow, Glasgow, UK 2 Wellcome Surgical Institute, University of Glasgow, Glasgow, UK 3 Glaxo Wellcome Research and Development, North Carolina, USA) ,  McCulloch, James (1 Department of Neuropathology, University of Glasgow, Glasgow, UK 2 Wellcome Surgical Institute, University of Glasgow, Glasgow, UK 3 Glaxo Wellcome Research and Development, North Carolina, USA) ,  Nilsen, Margaret (1 Department of Neuropathology, University of Glasgow, Glasgow, UK 2 Wellcome Surgical Institute, University of Glasgow, Glasgow, UK 3 Glaxo Wellcome Research and Development, North Carolina, USA) ,  Roses, Allen D. (1 Department of Neuropathology, University of Glasgow, Glasgow, UK 2 Wellcome Surgical Institute, University of Glasgow, Glasgow, UK 3 Glaxo Wellcome Research and Development, North Carolina, USA) ,  Nicoll, James A. R.

Abstract AI-Helper 아이콘AI-Helper

AbstractApolipoprotein E (apoE, protein; APOE, gene) is expressed as three isoforms in humans (E2, E3, E4). The APOE‐ε4 allele is associated with a poor outcome in patients after head injury of which ischaemic brain damage is a contributor of mortality and morbidity. The aim of the stu...

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참고문헌 (43)

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