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NTIS 바로가기Molecular psychiatry, v.6 no.2, 2001년, pp.230 - 234
Basile, V S (Neurogenetics Section, Clarke Institute of Psychiatry, Centre for Addiction and Mental Health (CAMH), University of Toronto, Toronto, Canada) , Ozdemir, V (Neurogenetics Section, Clarke Institute of Psychiatry, Centre for Addiction and Mental Health (CAMH), University of Toronto, Toronto, Canada, and Department of Pharmacology, University of Toronto, Toronto, Canada) , Masellis, M (Neurogenetics Section, Clarke Institute of Psychiatry, Centre for Addiction and Mental Health (CAMH), University of Toronto, Toronto, Canada) , Meltzer, H Y (Department of Psychiatry, Vanderbilt University, Nashville, USA) , Lieberman, J A (Department of Psychiatry, University of North Carolina, Chapel Hill, USA) , Potkin, S G (Department of Psychiatry, University of California, Irvine, USA) , Macciardi, F M (Neurogenetics Section, Clarke Institute of Psychiatry, Centre for Addiction and Mental Health (CAMH), University of Toronto, Toronto, Canada) , Petronis, A (Neurogenetics Section, Clarke Institute of Psychiatry, Centre for Addiction and Mental Health (CAMH), University of Toronto, Toronto, Canada) , Kennedy, J L (Neurog)
Tardive dyskinesia (TD) is a disabling neurological side effect associated with long-term treatment with typical antipsychotics. Family studies and animal models lend evidence for hereditary predisposition to TD. The newer atypical antipsychotics pose a minimal risk for TD which is in part attribute...
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