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NTIS 바로가기Kidney international, v.77 no.12, 2010년, pp.1060 - 1062
Satoh, Minoru (Division of Nephrology, Department of Internal Medicine, Kawasaki Medical School, Kurashiki, Japan)
The intrarenal renin–angiotensin system and reactive oxygen species contribute to the progression of chronic kidney disease. Godin et al. applied transgenic methods to prove renoprotection by catalase. They show that all of the pathological effects of overexpression of angiotensinogen can be a...
Kidney Int Godin 77 1086 2010 10.1038/ki.2010.63 Catalase overexpression prevents hypertension and tubular apoptosis in angiotensinogen transgenic mice
Kidney Int Brezniceanu 71 912 2007 10.1038/sj.ki.5002188 Catalase overexpression attenuates angiotensinogen expression and apoptosis in diabetic mice
Kidney Int Sachetelli 69 1016 2006 10.1038/sj.ki.5000210 RAS blockade decreases blood pressure and proteinuria in transgenic mice overexpressing rat angiotensinogen gene in the kidney
Kidney Int Liu 75 156 2009 10.1038/ki.2008.509 Apocynin attenuates tubular apoptosis and tubulointerstitial fibrosis in transgenic mice independent of hypertension
Kidney Int Kobayashi 68 1018 2005 10.1111/j.1523-1755.2005.00494.x Catalase deficiency renders remnant kidneys more susceptible to oxidant tissue injury and renal fibrosis in mice
J Am Soc Nephrol Yamamoto 18 1558 2007 10.1681/ASN.2006060554 Urinary angiotensinogen as a marker of intrarenal angiotensin II activity associated with deterioration of renal function in patients with chronic kidney disease
Hypertension Ogawa 47 699 2006 10.1161/01.HYP.0000203826.15076.4b Angiotensin II type 1 receptor blockers reduce urinary oxidative stress markers in hypertensive diabetic nephropathy
Biochem Biophys Res Commun Kobori 358 156 2007 10.1016/j.bbrc.2007.04.105 Enhanced intrarenal oxidative stress and angiotensinogen in IgA nephropathy patients
Exp Physiol Soler 93 549 2008 10.1113/expphysiol.2007.041350 Angiotensin-converting enzyme 2 and the kidney
J Am Soc Nephrol Nangaku 17 17 2006 10.1681/ASN.2005070757 Chronic hypoxia and tubulointerstitial injury: a final common pathway to end-stage renal failure
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