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NTIS 바로가기Cerebral cortex, v.25 no.2, 2015년, pp.482 - 495
Jantzie, Lauren L. (Department of Neurology, Boston Children's Hospital, Boston, MA 02115, USA) , Talos, Delia M. (Department of Neurology, Boston Children's Hospital, Boston, MA 02115, USA) , Jackson, Michele C. (Department of Neurology, Boston Children's Hospital, Boston, MA 02115, USA) , Park, Hyun-Kyung (Department of Neurology, Boston Children's Hospital, Boston, MA 02115, USA) , Graham, Dionne A. (Harvard Medical School, Boston, MA 02115, USA) , Lechpammer, Mirna (Department of Neurology, Boston Children's Hospital, Boston, MA 02115, USA) , Folkerth, Rebecca D. (Department of Neurology, Boston Children's Hospital, Boston, MA 02115, USA) , Volpe, Joseph J. (Department of Neurology, Boston Children's Hospital, Boston, MA 02115, USA) , Jensen, Frances E. (Department of Neurology, Boston Children's Hospital, Boston, MA 02115, USA)
The pathophysiology of perinatal brain injury is multifactorial and involves hypoxia-ischemia (HI) and inflammation. N-methyl-d-aspartate receptors (NMDAR) are present on neurons and glia in immature rodents, and NMDAR antagonists are protective in HI models. To enhance clinical translation of roden...
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