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Cell reports, v.17 no.1, 2016년, pp.165 - 178
Yao, Kai (Department of Ophthalmology and Visual Science, Yale University School of Medicine, New Haven, CT 06511, USA) , Qiu, Suo (Department of Ophthalmology and Visual Science, Yale University School of Medicine, New Haven, CT 06511, USA) , Tian, Lin (Department of Biochemistry and Molecular Medicine, School of Medicine, University of California, Davis, Davis, CA 95616, USA) , Snider, William D. (UNC Neuroscience Center, University of North Carolina, Chapel Hill, Chapel Hill, NC 27599, USA) , Flannery, John G. (Helen Wills Neuroscience Institute, University of California, Berkeley, Berkeley, CA 94720, USA) , Schaffer, David V. (Helen Wills Neuroscience Institute, University of California, Berkeley, Berkeley, CA 94720, USA) , Chen, Bo (Department of Ophthalmology and Visual Science, Yale University School of Medicine, New Haven, CT 06511, USA)
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Summary In cold-blooded vertebrates such as zebrafish, Müller glial cells (MGs) readily proliferate to replenish lost retinal neurons. In mammals, however, MGs lack regenerative capability as they do not spontaneously re-enter the cell cycle unless the retina is injured. Here, we show that gen...
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