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Biochemical and biophysical research communications, v.470 no.1, 2016년, pp.15 - 21
Achiwa, Koichi (Department of Gastroenterology and Hepatology, Nagoya University Graduate, School of Medicine) , Ishigami, Masatoshi (Department of Gastroenterology and Hepatology, Nagoya University Graduate, School of Medicine) , Ishizu, Yoji (Department of Gastroenterology and Hepatology, Nagoya University Graduate, School of Medicine) , Kuzuya, Teiji (Department of Gastroenterology and Hepatology, Nagoya University Graduate, School of Medicine) , Honda, Takashi (Department of Gastroenterology and Hepatology, Nagoya University Graduate, School of Medicine) , Hayashi, Kazuhiko (Department of Gastroenterology and Hepatology, Nagoya University Graduate, School of Medicine) , Hirooka, Yoshiki (Department of Gastroenterology and Hepatology, Nagoya University Graduate, School of Medicine) , Katano, Yoshiaki (Department of Internal Medicine, Banbuntane Hotokukai Hospital, Fujita Health University, School of Medicine) , Goto, Hidemi (Department of Gas)
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Abstract Nonalcoholic steatohepatitis (NASH) patients progress to liver cirrhosis and even hepatocellular carcinoma (HCC). Several lines of evidence indicate that accumulation of lipopolysaccharide (LPS) and disruption of gut microbiota play contributory roles in HCC. Moreover, in a dextran sodium ...
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